Human FUT3/CD174/FT3BORF/cDNA clone-Lentivirus plasmid (NM_000149)

SKU: pGMLV000456
Size: 10 µg
Leading Time: 3-7 working days

Pre-made Human FUT3/CD174/FT3B Lentiviral expression plasmid for FUT3 lentivirus packaging, FUT3 lentivirus production, overexpression stable cell line development, cell transient transfection and gene delivery targeting T/B/NK cells, macrophages, cardiomyocytes, hepatocytes, and neurons.

Our GM-Lentiviral vector is seamlessly integrated with the GM lentivirus packaging system. Discover more about the GM lentivirus packaging system.

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Product Description

Catalog ID pGMLV000456
Gene Name FUT3
Accession Number NM_000149
Gene ID 2525
Species Human
Product Type Lentivirus plasmid (overexpression)
Insert Length 1086 bp
Gene Alias CD174,FT3B,FucT-III,LE,Les
Fluorescent Reporter Firefly luciferase
Mammalian Cell Selection Puromyocin
Fusion Tag 3xflag (C-Terminal)
Promoter CMV
Resistance Amplicin

Reference




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    Associated products


    Category Cat No. Products Name
    Target Antibody GM-Tg-g-IP0076-Ab Anti-FUT3 monoclonal antibody
    Target Antigen GM-Tg-g-IP0076-Ag FUT3 protein
    ORF Viral Vector pGMLP003593 Human FUT3 Lentivirus plasmid
    ORF Viral Vector pGMLV000456 Human FUT3 Lentivirus plasmid
    ORF Viral Vector vGMLP003593 Human FUT3 Lentivirus particle
    ORF Viral Vector vGMLV000456 Human FUT3 Lentivirus particle


    Target information

    Target ID GM-IP0076
    Target Name FUT3
    Gene ID 2525
    Gene Symbol and Synonyms CD174,FT3B,FucT-III,FUT3,LE,Les
    Uniprot Accession P21217
    Uniprot Entry Name FUT3_HUMAN
    Protein Sub-location Introcelluar Protein
    Category Therapeutics Target, Immuno-oncology Target
    Disease Not Available
    Gene Ensembl ENSG00000171124
    Target Classification Checkpoint-Immuno Oncology

    The Lewis histo-blood group system comprises a set of fucosylated glycosphingolipids that are synthesized by exocrine epithelial cells and circulate in body fluids. The glycosphingolipids function in embryogenesis, tissue differentiation, tumor metastasis, inflammation, and bacterial adhesion. They are secondarily absorbed to red blood cells giving rise to their Lewis phenotype. This gene is a member of the fucosyltransferase family, which catalyzes the addition of fucose to precursor polysaccharides in the last step of Lewis antigen biosynthesis. It encodes an enzyme with alpha(1,3)-fucosyltransferase and alpha(1,4)-fucosyltransferase activities. Mutations in this gene are responsible for the majority of Lewis antigen-negative phenotypes. Differences in the expression of this gene are associated with host susceptibility to viral infection. [provided by RefSeq, Aug 2020]



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